This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Citing articles on Web of Science (14)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Study of the Mechanism

Mordecai M. Popovtzer, MD; Fred H. Katz, MD; Wulf F. Pinggera, MD; John Robinette; Charles G. Halgrimson, MD; Donald E. Butkus, MD

Arch Intern Med. 1973;132(2):203-208.

Abstract
Episodes of hyperkalemia associated with severe muscle weakness dominated the clinical course of a patient with salt-losing nephropathy. The evaluation of urinary excretory patterns of electrolytes under varying intakes of sodium suggested that a reduction of glomerular filtration below a critically low rate was the main factor responsible for renal retention of potassium with resulting hyperkalemia.

Author Affiliations
Denver

From the departments of medicine (Drs. Popovtzer, Katz, Pinggera, and Butkus and Mr. Robinette) and surgery (Dr. Halgrimson), University of Colorado Medical Center and Veterans Administration Hospital, Denver.

Footnotes
Received for publication Dec 14,1971; accepted July 7, 1972.

Reprint requests to 4200 E Ninth Ave, Denver 80220 (Dr. Popovtzer).

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?

THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Combined Tubular Dysfunction in Medullary Cystic Disease
Chagnac et al.
Arch Intern Med 1986;146:1007-1009.
ABSTRACT