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Vinod K. Bansal, MD; Marion H. Brooks, MD; John C. York, MD; Jessie E. Hano, MD

Arch Intern Med. 1979;139(1):100-102.

Abstract
A patient with chronic renal failure developed intractable hypoglycemia after 2 1/2 years of dialysis. Metabolic studies did not suggest that malnutrition, substrate limitation, hormone deficiency, or insulin excess were responsible for the low blood glucose level. Impaired gluconeogenesis may have been an important factor, but studies in this patient suggest that enhanced glucose utilization may also play a role in the pathogenesis of hypoglycemia in renal failure.

(Arch Intern Med 139:100-102, 1979)

Author Affiliations
From the Departments of Medicine (Drs Bansal, Brooks, and Hano) and Pathology (Dr York), Loyola University Medical Center, Maywood, Ill.

Footnotes
Accepted for publication June 26, 1978.

Reprint requests to Department of Medicine, Loyola University Medical Center, 2160 S First Ave, Maywood, IL 60153 (Dr Brooks).

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