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The administration of metoclopramide hydrochloride is commonly used as a provocative test in the screening of patients with pheochromocytoma because it produces a severe pressor reaction in these patients.¹ This phenomenon has been attributed to catecholamine-releasing effects of the antidopaminergic agent.¹ However, a dissociation between blood pressure (BP) and plasma catecholamines has occurred in patients with pheochromocytoma,² suggesting that other endocrine factors may play a role in the control of BP in these patients. Specifically, the pressor response during stimulation with metoclopramide might be mediated at least in part by enhanced circulating levels of vasopressin; the stimulatory effect of metoclopramide on vasopressin secretion is well known.³ To verify this possibility, we have evaluated the arginine vasopressin (AVP) secretory pattern in 3 patients affected by pheochromocytoma during stimulation with metoclopramide.

Report of Cases

Case 1

A 47-year-old woman was admitted to the hospital with crises of hypertension (BP, 190/110 mm Hg), tachycardia (heart rate, 110 beats/min), . . . [Full Text of this Article]

Case 2

Case 3

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