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It was intriguing going through the clinical observations of Kahn¹ concerning patients with hypernatremia in association with edema. He described 7 hospitalized patients with hypoalbuminemia and azotemia presenting with increased serum sodium levels and marked edema.¹ In this context, hypernatremia indicates sodium excess in the presence of fluid excess. Furthermore, urine output was increased, implying defective water conservation induced by a lower than expected level of antidiuretic hormone (ADH) activity caused 7y hypernatremia.¹

We recently studied a number of hospitalized patients with impaired mental status who developed hypernatremia without marked edema. These patients were divided into 2 groups. The first group consisted of 4 patients who were admitted because of malignant neoplasm–related hypercalcemia. In this setting, isotonic sodium chloride solution together with potassium supplements was administered intravenously to replenish intravascular volume and restore calcium and potassium levels. The second group consisted of 5 patients who experienced stroke and who received . . . [Full Text of this Article]

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