This Article  • Full text  • PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on Web of Science (1)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Arch Intern Med. 2001;161:291-292.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Hyponatremia is a common adverse effect of many drugs (diuretics, antipsychotic agents, carbamazepine, and others). Among the uncommon causes of hyponatremia are theophylline and trimethoprim. We describe a woman who presented with severe symptomatic hyponatremia following treatment with anhydrous theophylline and developed trimethoprim-induced severe hyponatremia 6 months later.

Report of a Case

A 78-year-old woman was admitted to the hospital because of severe hyponatremia (plasma sodium level, 117 mmol/L), weakness, abdominal pain, nausea, and vomiting. Her medical history included arterial hypertension, hypercholesterolemia, bronchial asthma, occasional abdominal pain, and dizziness, for which she was treated with cilazapril, albuterol sulfate, budesonide, famotidine, and mebeverine hydrochloride. One week before admission, she was treated with trimethoprim-sulfamethoxazole for a presumptive urinary tract infection.

Six months before her admission, the patient had been hospitalized in another ward because of abdominal pain, nausea, weakness, tachycardia, and hyponatremia (plasma sodium level, 122 mmol/L). Her medications at that time included theophylline, amlodipine besylate, . . . [Full Text of this Article]

Comment


CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?