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Arch Intern Med. 2001;161:1114-1115.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Diabetes insipidus (DI), which is characterized by polyuria and polydipsia due to excessive urinary loss of solute-free water, can be either central (CDI) or nephrogenic.¹ In CDI, plasma levels of arginine vasopressin do not increase in response to a rise in plasma osmolarity. A number of intracranial conditions, such as neoplastic or infiltrative lesions of the hypothalamus or pituitary gland, severe head injuries, neurosurgery, and central nervous system infection, can cause CDI.¹ Central DI has been reported as a complication of bacterial meningitis, but most of the cases have occurred in children.² We describe a 45-year-old man with human immunodeficiency virus (HIV) infection and Streptococcus pneumoniae meningitis who developed central diabetes insipidus, most likely as a complication of raised intracranial pressure.

Report of a Case

A 45-year-old man was admitted to the hospital with headaches, fever, and delirium. His medical history included HIV infection, which had been diagnosed 2 months before admission. His CD4 . . . [Full Text of this Article]

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THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

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