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Arch Intern Med. 2005;165:2058-2060.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Considerable media attention has focused on recent research reports that statin-associated levels of C-reactive protein (CRP) are associated with prognosis after acute coronary syndrome.^1-2 Similar media attention to this topic is not new: the "CRP story" and reports of inflammation in general as related to heart disease have been repeatedly featured over the past several years in major print magazines, newspapers, and television news magazines. However, the most recent attention focused on this topic, including the relatively new phenomenon of "recommendations" made by popular media editorialists, raises 3 important concerns, 1 of which is elucidated by Miller et al³ in this issue of the ARCHIVES.

WHAT ARE CRP AND "HIGH-SENSITIVITY CRP" AND HOW DO THEY RELATE TO ATHEROSCLEROSIS?

The term C-reactive protein was coined in 1930 by Tillet and Frances, who first identified this protein as reactive with the "C" component of the pneumococcus bacterial coat.⁴ Since that time, evaluation of CRP level has become crucial to monitor inflammation such as . . . [Full Text of this Article]

DOES CRP REPRESENT A FUNDAMENTALLY NEW WAY TO VIEW HEART DISEASE?

WHEN SHOULD CRP BE MEASURED?

SHOULD CRP BE A SPECIFIC THERAPEUTIC TARGET?

AUTHOR INFORMATION
Russell P. Tracy, PhD; Lewis H. Kuller, MD, MPH

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