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Spencer B. King III, MD

Arch Intern Med. 2005;165:2589-2592.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

INTRODUCTION

Therapy for ischemic heart disease has the potential for improving 2 types of outcomes: hard events such as death and myocardial infarction or symptoms. Evidence for prolongation of life comes by inference from similar conditions that have been treated surgically. Whereas percutaneous coronary intervention (PCI) is certainly lifesaving in some subsets, a recent meta-analysis of 11 small controlled trials did not show improved survival.¹ Various medical therapies have shown benefit in avoiding death and myocardial infarction separately and in combination. Such effective therapies should always be used to minimize these events. This article, therefore, will concentrate on the value of PCI to relieve the symptoms of chronic angina pectoris.

Angina is believed to be caused by the release of chemical substances, including kinins, serotonin, hydrogen ions, and other factors that are produced in an environment of ischemia. These agents stimulate nerve endings in the heart, . . . [Full Text of this Article]

IS PCI SUPERIOR TO MEDICAL THERAPY ALONE FOR ANGINA PECTORIS?

TRIALS COMPARING STENTING TO OPTIMAL MEDICAL THERAPY

SEVERELY SYMPTOMATIC PATIENTS

EVIDENCE FROM ACUTE CORONARY SYNDROME TRIALS

IMPACT OF DRUG-ELUTING STENTS

LIMITATIONS

AUTHOR INFORMATION

Author Affiliations: Fuqua Heart Center, Piedmont Hospital, Atlanta, Ga.

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