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Niskanen and colleagues¹ recently demonstrated that uric acid (UA) levels were a significant independent predictor of cardiovascular disease (CVD) mortality in healthy middle-aged men after comprehensive assessment and adjustment for variables associated with the gout or metabolic syndrome. However, they do not address the individual categories of alcoholic beverages, the incident cases of gout during the long-term follow-up, and the effect of initiating UA-lowering pharmacotherapy for men at the second tertile (UA level, 5.05-5.88 mg/dL [300.37-349.74 µmol/L]), all of whom had normal UA levels below 7 mg/dL (<416.36 µmol/L).²

In earlier years, it has been reported that the high purine content in beer might play a leading role in the risk of gout.³ Furthermore, Choi et al⁴ had recently indicated that beer consumption revealed the strongest association with the risk of gout, and spirits intake was significantly relevant to the event as well. Therefore, we wonder whether adjustment for different . . . [Full Text of this Article]

AUTHOR INFORMATION
Shih-Chen Kuo, RPh, MSCP; Yea-Huei Kao Yang, RPh, BSPharm

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