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Peter Lee, MBBS(Hons); Graham R. D. Jones, PhD; Jacqueline R. Center, MBBS, FRACP, PhD

Arch Intern Med. 2008;168(3):325-326.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Hyponatremia following cerebral trauma has commonly been attributed to the syndrome of inappropriate secretion of antidiuretic hormone (SIADH). Cerebral salt wasting (CSW) can lead to a similar clinical picture, for which treatment is not well defined.

Report of a Case. A 75-year-old man presented with a 3-week history of worsening ataxia following blunt head trauma in a motor vehicle crash 1 month earlier. His history included uncomplicated type 2 diabetes mellitus and hypertension, managed with rosiglitazone, 4 mg/d, and ramipril, 2.5 mg/d. The patient was confused and had marked truncal ataxia. Central venous pressure was low at 3 mm Hg, indicating volume depletion. Euglycemia and normotension were maintained following the discontinuation of both rosiglitazone and ramipril therapies.

A computed tomographic scan of the brain demonstrated acute and chronic bilateral frontoparietal subdural hematomas measuring up to 22 . . . [Full Text of this Article]

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