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HOWARD H. HIATT, M.D.; SAMUEL LOWIS, M.D.

AMA Arch Intern Med. 1957;100(1):143-146.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The paradox of hyperosmolarity of the extracellular fluid and reduced urinary excretion of sodium and chloride is frequently encountered in persons who have suffered head trauma. We have recently successfully treated this complication with posterior pituitary extract in a patient who had sustained a severe head injury. Evidence is presented to suggest that the electrolyte disturbance was the result of the development of diabetes insipidus. Diabetes insipidus resulting in hypernatremia in a patient with brain damage was first discussed by Peters.¹

Report of Case

A 39-year-old comatose male painter was transferred to the Beth Israel Hospital from the Hale Hospital, Haverhill, Mass., on Jan. 28, 1956. Eight days previously he had fallen 14 ft. from a scaffolding and struck the right frontal portion of his skull on a concrete surface. He had been unconscious from the time of the fall. Examination at the Hale Hospital had revealed bleeding from . . . [Full Text PDF of this Article]

Author Affiliations
Boston

From the Departments of Medicine and Surgery, Beth Israel Hospital and Harvard Medical School.

Footnotes
Received for publication Sept. 11, 1956.

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