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Height, Weight, and Their Ratio in the Accelerated Form of Primary Hypertension
GEORGE A. PERERA, M.D.;
ALBERT DAMON, M.D.
AMA Arch Intern Med. 1957;100(2):263-265.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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The accelerated ("malignant") form of hypertension is characterized by the presence of pathological changes indicative of a widespread necrotizing arteriolitis. Strong but not certain evidence for this diagnosis may be obtained on clinical grounds, particularly when patients are encountered with established diastolic hypertension, with rapidly progressive renal damage, and usually—although not necessarily—with retinopathy and papilledema.
Separation of the accelerated form from other types of hypertension was made by Volhard and Fahr, in 1914.1 Since that time there have been numerous descriptions of its clinical features and its rather uniform rapidly downhill course.2-5 It is accepted generally that this condition occurs infrequently de novo, appearing more often after the previous development of primary (essential) or secondary hypertension.
Discussion continues as to whether one is dealing with a phase dependent solely upon the intensity of the hypertensive process or whether a superimposed and qualitatively different mechanism is responsible.6 The
. . . [Full Text PDF of this Article]
Author Affiliations
New York
From the Department of Medicine, Columbia University College of Physicians and Surgeons, and the Presbyterian Hospital. Fellow of the American Heart Association (Dr. Damon).
Footnotes
Accepted for publication March 19, 1957.
This study was supported by grants from the National Heart Institute (U. S. P. H. S.), the Union County Heart Association (New Jersey), the Albert and Mary Lasker Foundation, the Albert H. and Jessie D. Wiggin Foundation, and the Irwin Strasburger Memorial Medical Foundation.
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