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HALCOTT T. HADEN, M.D.

AMA Arch Intern Med. 1957;100(6):986-988.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Vitamin K deficiency as the cause of a hemorrhagic diathesis has been recognized for many years. Clinical deficiency of this vitamin has usually been associated with some defect in absorption from the gastrointestinal tract, such as that associated with biliary-tract obstruction or with sprue. Dietary deficiency of vitamin K has not been important because of the ability of the normal intestinal bacteria to synthesize vitamin K. Since the introduction of the broadspectrum antibiotics it has become possible to greatly reduce the bacterial content of the intestine and, as a consequence, to decrease the amount of vitamin K produced. The fact that this can occasionally produce a severe hemorrhagic diathesis is attested by the following case.

Report of Case

A 46-year-old white woman was admitted to the urology service of the Medical College of Virginia Hospital on June 6, 1956, because of right ureteral obstruction and chronic renal infection. Five years . . . [Full Text PDF of this Article]

Author Affiliations
Atlanta

Footnotes
Submitted for publication May 22, 1957.

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