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  Vol. 101 No. 4, APRIL 1958 TABLE OF CONTENTS
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Studies in Headache Mechanisms

ADRIAN M. OSTFELD, M.D.; HELEN GOODELL, B.S.; HAROLD G. WOLFF, M.D.

AMA Arch Intern Med. 1958;101(4):755-760.

Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Introduction

Headache may be the commonest complaint heard in physicians' offices. The great majority of headaches are linked either with cranial vasodilatation or skeletal muscle contraction and cranial vasoconstriction. The former are called migraine; the latter, muscle-contraction headache.1 Changes in gastric function, feeling state, nasal function, fluid and electrolyte excretion, cardiac rhythm, and body temperature regulation may occur in association with headache

Fig. 1.—Cranial artery pulse wave recorder. attack. A part of our newer knowledge of migraine headache phenomenology is herein reviewed.

Fig. 2.—Poser slit lamp and camera attachment for study of bulbar conjunctival vessels.

Headache mechanisms were studied with use of the devices shown in Figures 1 and 2.

Fig. 3.—Effect of topical acetylcholine on bulbar conjunctival vessels. Contrast the normal vessels at the top with the minimal dilatation (sensitivity) at bottom.

Fig. 4.—Effect of topical levarterenol on bulbar conjunctival vessels. Those at the top are normal. At . . . [Full Text PDF of this Article]


Author Affiliations

Chicago; New York

From the study program in Human Health and the Ecology of Man and the Departments of Medicine (Neurology) and Psychiatry, Cornell University Medical College. Present address of Dr. Ostfeld: University of Illinois College of Medicine.


Footnotes

Submitted for publication Nov. 7, 1956.

Shown as a scientific exhibit of the Section on Internal Medicine at the 105th Annual Meeting of the American Medical Association, Chicago, June 11-15, 1956.



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