This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Citing articles on Web of Science (39)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

CAPT. CHARLES R. BAXTER, MC; MAJOR PAUL E. TESCHAN, MC

AMA Arch Intern Med. 1958;101(6):1040-1050.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The criteria for the diagnosis of heat stroke are well known, but atypical clinical findings, such as abnormal behavior patterns without severe hyperpyrexia following exposure to heat, may go unrecognized.¹ Detailed chemical studies are lacking in all reported cases of heat injury. A small but definite incidence of acute renal failure occurring in the course of severe heat injury has been reported.¹,¹⁶,¹⁸ However, relatively little clinical and chemical detail has been included.

Three fatal cases of heat injury with oliguria occurring in military personnel stationed near San Antonio, Texas, are presented to (1) describe some clinical and chemical findings that may precede this atypical clinical form of heat stroke, (2) suggest means of preventing the associated acute renal failure with oliguria, and (3) account for the deaths of these patients on the basis of shock, persistent hyperthermia, and remarkably rapid rise in plasma potassium concentration with . . . [Full Text PDF of this Article]

Author Affiliations
U. S. Army

From the U. S. Army Surgical Research Unit, Brooke Army Medical Center, Fort Sam Houston, Texas.

Footnotes
Submitted for publication Nov. 4, 1957.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?