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HABEEB BACCHUS, Ph.D., M.D.

AMA Arch Intern Med. 1958;102(2):194-198.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

It is becoming increasingly clear that cerebrovascular hemodynamics are influenced by alterations in cardiopulmonary physiology. The work of Kety and Schmidt¹ revealed that an increased cerebral blood flow occurs in hypercapnia and in hypoxia. It has also been shown that the increased cerebral blood flow in such situations is followed by increased cerebrospinal fluid pressure.² The exact mechanism whereby these changes influence cerebral vascular dynamics is not clear. Clinical syndromes attesting the relationship between cardiopulmonary physiology and cerebrovascular dynamics have been described. Within the past two decades reports have appeared demonstrating signs and symptoms of intracranial disease in patients with primary pulmonary problems. These reports have been well reviewed by Conn et al.,³ who also added two cases of their own of patients with "emphysematous encephalopathy."

In recent months we have had the opportunity of studying two patients with pulmonary disease associated with clinical findings suggesting the . . . [Full Text PDF of this Article]

Author Affiliations
Bethesda, Md.

Chief Resident, Department of Medicine, Providence Hospital, Washington D. C. Present address: Metabolism Service, National Cancer Institute, National Institutes of Health, Bethesda, Md.

Footnotes
Submitted for publication Nov. 19, 1957.

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