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V. GILSANZ, M.D.; M. GALLEGO, M.D.; A. VERGARA, M.D.

AMA Arch Intern Med. 1960;105(2):252-256.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

It seemed of interest to us to study the portal circulation in patients with myxedema. A great deal has already been published on circulatory alterations in hypothyrodism, especially in the so-called myxedematous heart. Hypothyroidism should in principle affect the circulation of all parts, but the fact that the liver in myxedema is habitually enlarged (be it due to edema or excessive deposition of glycogen, etc.) makes us wonder if at the same time there are alterations in the portal circulation and if so, whether this is just a reflection of the general circulatory disturbance in hypothyroidism or perhaps caused by some local factor that could explain it.

Our investigations were thus oriented along the following lines:

1. Examination of hepatic biopsies to ascertain any alterations in the myxedematous liver, and in case any were found to see if they are reversible upon treatment.
   
2. Determination of the intrasplenic venous pressure (which is
   

Author Affiliations
Madrid, Spain

Department of Internal Medicine, University of Madrid (Prof. Dr. V. Gilsanz).

Footnotes
Submitted for publication April 14, 1959.

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