This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

CAPT. ARTHUR S. WEISSBEIN, MC; LT. COL. NORMAN M. SCOTT, Jr., MC

Arch Intern Med. 1960;106(1):71-80.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Postnecrotic cirrhosis is considered, in the United States, almost always to be a residual of infectious or homologous serum hepatitis, although in occasional instances, exposure to toxic chemicals may be blamed.¹ In the tropics, malnutrition is believed to be an important etiologic factor. Whatever the basic cause, all appear to have in common the ability to produce the diffuse hepatocellular necrosis required in the pathogenesis of postnecrotic cirrhosis.

During and since World War II, there has been a high incidence of hepatitis throughout the world. This has stimulated great interest in the possible residuals of that disease. In our experience and in that of others,^2,3 there also has recently been an increase in the frequency of postnecrotic cirrhosis, and it is natural to associate the two conditions. There have been, however, a number of patients in whom postnecrotic cirrhosis appears to have developed de novo.^4,5 Not only . . . [Full Text PDF of this Article]

Author Affiliations
U.S. Army

Footnotes
Submitted for publication Sept. 28, 1959.

Senior Resident, Internal Medicine (Capt. Weissbein), and Chief, Gastroenterology Service (Lt. Col. Scott), Brooke Army Hospital, Brooke Army Medical Center, Fort Sam Houston, Texas.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?