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Part III. The Mechanism of Pain; Based on a Clinicopathological Investigation of 100 Cases of Minor and 100 Cases of Massive Embolism of the Pulmonary Artery

L. WHITTINGTON GORHAM, M.D.

Arch Intern Med. 1961;108(3):418-426.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

A number of theories have been advanced to explain the cause of the two types of pain, pleural and anginal, which occur in embolism of the pulmonary artery. A discussion of these has assumed increasing importance in recent years, because pulmonary embolism has now become the most frequently encountered lesion of the lung in general hospitals,¹ since modern drug and antibiotic therapy have greatly reduced the incidence of infectious diseases of the lung. The clinicopathological observations here recorded deal with the mechanisms involved in the two types of pain. The data were obtained from a study of 385 patients autopsied at the New York Hospital. Of these, 100 were instances of massive pulmonary embolism (A Study of Pulmonary Embolism: Part I, Table 1), with large emboli in the main stem, the two major branches, or in a single major branch, while 100 were examples of medium-sized smaller emboli occluding . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Department of Pathology, New York Hospital-Cornell Medical Center.; Research Associate in the Department of Pathology; Research Consultant, Goldwater Memorial Hospital, First Research Service of the College of Physicians and Surgeons, Columbia University; Professor of Medicine (Emeritus), Albany Medical College of Union University; Director (Emeritus) of the Public Health Research Institute of the City of New York, Inc.

Footnotes
Submitted for publication Aug. 19, 1960.

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