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Postpartal Heart Disease
John J. Walsh, M.D.;
George E. Burch, M.D.
Arch Intern Med. 1961;108(6):817-822.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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To the practicing physician and to the research investigator alike, the dilated heart has long occupied an enigmatic but challenging role. Transcending etiological diagnosis, ubiquitous to the extent of being commonplace to the pathologist, cardiac dilation, nevertheless, is a major determinant of all functional and therapeutic classification. Even the tempo of the clinical course may be set by the extent to which the heart has become dilated. It follows, then, that in an attempt to elucidate the mechanisms operative in the development of cardiac enlargement, so as better to reverse this process, attention should be directed to that group of myocardial diseases termed myocardoses and myocardites. In many instances, these entities appear to represent a single insult to the myocardium, in contradistinction to the continuing noxious stress of hypertension, congenital defects, valvular deformity, and coronary arterial insufficiency. Even in the case of protracted disturbances, e.g. malnutrition, the areas of disturbed
. . . [Full Text PDF of this Article]
Author Affiliations
Tulane University School of Medicine 1430 Tulane Ave., New Orleans 12.
From the Cardiovascular Research Laboratory of the U.S. Public Health Service Hospital, the Charity Hospital of Louisiana, and the Tulane Medical School, New Orleans, Louisiana.
Footnotes
Aided in part by U.S. Public Heath Service Grant H-4012.
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