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Hypertension Produced by Unilateral Renal Disease
JOHN EAGER HOWARD, M.D.;
THOMAS B. CONNOR, M.D.
Arch Intern Med. 1962;109(1):8-17.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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There is ample reason to believe that, in man, reduced arterial flow to one kidney may result in hypertension. This is evidenced by many reports of long-term reversion to normotension after nephrectomy,1-12 and by some more recently reported short-term results after plastic surgical procedures upon stenotic renal arteries.13-15 The underlying mechanism whereby hypertension is brought about in these situations, however, remains unknown.
In the period immediately following Butler's initial report of restoration of normotension after removal of a diseased kidney,16 many nephrectomies were performed, without benefit to the patients in most instances.17-19 The best results achieved in any of the larger series of cases disclosed that only 30% of the patients had improved. In the past decade further procedures have been devised to evaluate whether or not a morbid kidney is responsible for the hypertension present. It is believed by the authors that (1) proper utilization
. . . [Full Text PDF of this Article]
Author Affiliations
BALTIMORE
Professor of Medicine, The Johns Hopkins University (Dr. Howard); Associate Professor of Medicine, University of Maryland (Dr. Connor).
Footnotes
Submitted for publication Feb. 20, 1961.
This work is supported in part by Grant No. 2A-5058 from the National Institute of Arthritis and Metabolic Diseases, National Institutes of Health, U.S. Public Health Service.
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