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With Electromyographic Evaluation

ROBERT L. RAINEY, M.D.; PAUL W. ESTES, M.D.; CHARLES L. NEELY, M.D.; LAWRENCE D. AMICK, M.D

Arch Intern Med. 1963;111(5):564-571.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Myoglobinuria may result whenever dissolution of muscle sarcoplasm occurs. This phenomenon is classified as primary, a relatively rare disease of unknown etiology, or as secondary when precipitated by crush injury, arterial occlusion with ischemia, high-voltage electrical shock, severe burns, or ingestion by a susceptible person of a certain type of fish (Haff disease).

Primary myoglobinuria is probably more common than the approximate 58 reported cases suggest. Although usually precipitated by muscular exertion, such is not the case in a significant number of patients. Fever,¹ infection,² and electroshock therapy³ have been implicated as inducing this disorder. Korein et al.⁴ classify in a subgroup all cases of primary myoglobinuria not preceded by muscular exertion, designating this group idiopathic or toxic myoglobinuria.^*

This case is of interest because of its association with the therapy of diabetic acidosis, because it is the first case in which electromyographic studies were done . . . [Full Text PDF of this Article]

Author Affiliations
MEMPHIS

From the Department of Internal Medicine and of Medical Laboratories, College of Medicine, University of Tennessee, and the City of Memphis Hospitals.; Work done during tenure as a Research Fellow of the American Heart Association.

Footnotes
Received for publication May 14, 1962; accepted Sept. 18.

Preparation of this report supported in part by Public Health Service Research Grant H-5684 from the National Heart Institute.

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