You are seeing this message because your Web browser does not support basic Web standards. Find out more about why this message is appearing and what you can do to make your experience on this site better.


ABOUT ARCHIVES
Advanced Search

Welcome   | My Account | E-mail Alerts | Access Rights | Sign In


  Vol. 116 No. 1, July 1965 TABLE OF CONTENTS
  Archives
  •  Online Features
  ARTICLES
 This Article
 •References
 •Full text PDF
 •Send to a friend
 • Save in My Folder
 •Save to citation manager
 •Permissions
 Citing Articles
 •Citation map
 •Citing articles on HighWire
 •Citing articles on Web of Science (37)
 •Contact me when this article is cited
 Related Content
 •Similar articles in this journal
 Social Bookmarking
  Add to CiteULike Add to Connotea Add to Del.icio.us Add to Digg Add to Reddit Add to Technorati Add to Twitter What's this?

Cerebral Salt-Wasting Associated With the Guillain-Barré Syndrome

LT CDR WILLIAM C. COOPER, MC; LT CDR IRVING J. GREEN, MC; SAN-PIN WANG, MD

Arch Intern Med. 1965;116(1):113-119.

Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

INAPPROPRIATE secretion of antidiuretic hormone has been implicated as the cause of the hyponatremia and renal salt-wasting associated with certain cases of intracranial and intrathoracic disease. Cases of traumatic, infectious, and neoplastic intracranial disease, and of neoplastic thoracic disease have been associated with the salt-wasting syndrome.1-6 The following is a report of the simultaneous occurrence in a patient of an illness clinically indistinguishable from the Guillain-Barré syndrome and of an electrolyte disturbance marked by transient loss of renal sodium-conserving ability, plasma hypo-osmolality, and plasma hyponatremia. Immunologic features of this patient's illness suggested to us that the etiology of the polyradiculoneuropathy was hypersensitivity to viral antigens.

Report of a Case

A 32-year-old Taiwanese man, a virology technician at the US Naval Medical Research Unit No. 2 (NAMRU-2), was admitted to Taiwan University Hospital on Sept 16, 1961, with the chief complaint of walking disturbance and pain in the left . . . [Full Text PDF of this Article]


Author Affiliations

USN; USN; TAIPEI, TAIWAN, REPUBLIC OF CHINA

From the Clinical Investigation Department, US Naval Medical Research Unit No. 2 (NAMRU-2). Clinical investigator (Dr. Cooper) and virologist (Drs. Green and Wang). Present addresses: Department of Nutrition and Food Science, Massachusetts Institute of Technology, Cambridge, Mass (Dr. Cooper); University of California Medical Center, Los Angeles (Dr. Green); Department of Preventive Medicine, University of Washington School of Medicine, Seattle (Dr. Wang).


Footnotes

Received for publication Dec 18, 1964; accepted Jan 15, 1965.

Read before the First Asian and Oceanian Congress of Neurology, Tokyo, Oct 10, 1963.

The opinions and assertions contained herein are those of the authors and are not to be construed as official or reflecting the views of the Navy Department or the Naval Service at large.

Reprint requests to Department of Nutrition and Food Science, Massachusetts Institute of Technology, Cambridge, Mass 02139 (Dr. Cooper).



Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us   Add to Digg Digg   Add to Reddit Reddit   Add to Technorati Technorati   Add to Twitter Twitter     What's this?





HOME | CURRENT ISSUE | PAST ISSUES | TOPIC COLLECTIONS | CME | SUBMIT | SUBSCRIBE | HELP
CONDITIONS OF USE | PRIVACY POLICY | CONTACT US | SITE MAP
 
© 1965 American Medical Association. All Rights Reserved.