 |
|
Lipogenic HypercholesteremiaA Guide for Reorientation in the Consideration of Lipid-Cholesterol Relationships
MEYER FRIEDMAN, MD;
SANFORD O. BYERS, PhD;
RAY H. ROSENMAN, MD
Arch Intern Med. 1965;116(6):807-809.
|
|
| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
|
|
|
IN the period from 1955-1957, the experimental elevation of either the plasma phosphatide or triglyceride of the laboratory animal was observed 1-5 to induce a hypercholesteremic state. Previous to these findings, we, like most investigators in this field, considered the plasma excess of phosphatide, triglyceride, or both in various hypercholesteremic states as secondary to or compensator for an initial derangement in cholesterol metabolism. However after such observations had been made, we began to reexamine various common clinical hypercholesteremic states with the possibility in mind that perhaps the plasma excess of phosphatide and/or triglyceride was not a sequent of the hypercholesteremia but the latter's causant. Such reexaminations now have convinced us that in various hypercholesteremic states, the initial cause of the disorder is indeed a lipogenic one in that it has been caused or set in motion by a primary error in phosphatide or triglyceride metabolism. Some of the reasons leading
. . . [Full Text PDF of this Article]
Author Affiliations
SAN FRANCISCO
From the Harold Brunn Institute, Mount Zion Hospital and Medical Center, San Francisco.
Footnotes
Received for publication June 2, 1965; accepted Aug 2.
Reprint requests to the Brunn Institute, Mount Zion Hospital and Medical Center, San Francisco, Calif 94115 (Dr. Friedman).
 CiteULike  Connotea  Del.icio.us  Digg  Reddit  Technorati  Twitter
What's this?
|
