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RICHARD G. NEIDBALLA, MD

Arch Intern Med. 1966;117(1):54-56.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THE DIVERSION OF portal venous blood into the systemic circulation through naturally occurring collateral vessels has long been recognized as a concomitant of processes which produce portal hypertension. Many of these vessel systems are familiar to the clinician as the hemorrhoidal, esophageal, and anterior abdominal wall plexuses. These various plexuses might be termed the normal porta-systemic communications, in that they are believed to be normally patent and respond to increased portal pressure by dilatation.

Splenoportography has enabled clinicians to recognize a second group of collateral pathways which have been designated the embryonal porta-systemic communications.¹ Presumably, these are embryonic channels which are thought to be reopened or recanalized by the high pressures obtained in the portal system. Such pathways have been noted in approximately 14% of patients with portal hypertension in separate studies by Hamilton and Sullivan ¹ and by Rousellot et al.² These communications have most commonly been . . . [Full Text PDF of this Article]

Author Affiliations
MINNEAPOLIS

From the University Hospitals, University of Minnesota Medical School, and the Minneapolis Veterans Administration Hospital, Minneapolis.

Footnotes
Received for publication April 28, 1965; accepted Aug 20.

Read before the Minnesota Society of Internal Medicine Fall Meeting, Oct 19, 1964.

Reprint requests to Minneapolis Veterans Administration Hospital, 54th & 48th St, Minneapolis, Minn 55417.

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