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CHRISTIAN B. J. von HOYNINGEN-HUENE, MD

Arch Intern Med. 1966;118(5):471-477.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

GOUT in children is a rare disease and provides a challenge to demonstrate more clearly the basic cause of gout. The isolated case reports which appear in the literature, however, only underline its rarity and do not contribute significantly to a better understanding of the disease. More recently, reports have been published of hyperuricemia and gout appearing in one adolescent and several adults with glycogen storage disease of the von Gierke or glucose-6-phosphatase deficient type often called type I^1-8 and hereafter referred to as glycogenosis. Even if a study of these cases does not clarify the cause of primary gout it still illuminates some of the several factors promoting hyperuricemia and may point to the basic mechanism responsible for the development of gout in these patients.

The following is a report of gout occurring in two preadolescent male siblings with glycogenosis.

Report of Cases

CASE 1.

—Patient (Fig 1) . . . [Full Text PDF of this Article]

Author Affiliations
SEMBACH, GERMANY

From the 38th Tactical Hospital, Sembach, Germany. Dr. von Hoyningen-Huene is presently with the Lovelace Clinic, Albuquerque, NM.

Footnotes
Received for publication April 1, 1966; accepted July 9.

Reprint requests to the Lovelace Clinic, Albuquerque, NM (Dr. von Hoyningen-Huene).

The opinions or conclusions contained in this paper are those of the author and do not necessarily reflect the views or official policy of the US Air Force.

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