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Thomas Kahn, MD; Marvin F. Levitt, MD

Arch Intern Med. 1970;126(4):664-667.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

It has been known for many years that patients with chronic renal disease cannot conserve salt as well as normal individuals. However, in 1942 Thorn et al described two patients with chronic renal disease who required a daily intake of 10 to 15 gms of salt in order to maintain salt and water balance.¹ These "salt wasters" did not respond to the administration of desoxycorticosterone or antidiuretic hormone, and it was concluded that the salt loss was consequent to a primary failure of tubular reabsorption of sodium. About 20 cases of massive urinary salt loss and renal failure, usually of obscure etiology, have subsequently been described in the literature.

Two patients had multiple myeloma and heavy urinary sodium wasting, and both required an intake of more than 10 gm of salt a day in order to remain in sodium balance.

Patient Summaries

PATIENT 1.

—A 67-year-old man was admitted . . . [Full Text PDF of this Article]

Author Affiliations
New York

Footnotes
Received for publication Nov 20, 1969; accepted April 29, 1970.

From the Division of Renal Diseases, Department of Medicine, Mount Sinai School of Medicine of the City University of New York. Reprint requests to Division of Renal Diseases, Department of Medicine, Mount Sinai School of Medicine of the City University of New York, Fifth Avenue and 100th Street, New York 10029 (Dr. Kahn).

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