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Disseminated Intravascular Coagulation in Trypanosomiasis
Elizabeth Barrett-Connor, MD;
Richard J. Ugoretz, MD;
Abraham I. Braude, MD, PhD
Arch Intern Med. 1973;131(4):574-577.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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Coagulation factors may be depleted in many different infections.1-3 In this country their depletion results primarily from severe bacterial infection, but in other parts of the world, where parasitic diseases are more common, it is possible that clotting factors may frequently disappear from the blood of patients with protozoal infections, as suggested by reports of intravascular coagulation in malaria.4,5 We have also observed depletion of clotting factors in a patient with African trypanosomiasis. Because there are no published descriptions of this complication of trypanosomiasis, we are reporting it to warn others of this serious problem in protozoan infections.
Patient Summary
A 19-year-old student was admitted to University Hospital with a diagnosis of African trypanosomiasis. He was one of ten Californians on a hunting safari in East Africa arriving in Maun, Botswana (formerly Bechuanaland) on July 31, 1970. All were heavily bitten by tsetse flies and no sleeping sickness
. . . [Full Text PDF of this Article]
Author Affiliations
La Jolla, Calif
From the departments of community medicine and medicine and pathology, University of California, San Diego School of Medicine, La Jolla.
Footnotes
Received for publication Dec 13,1971; accepted March 7, 1972.
Reprint requests to Veterans Administration Hospital, 3350 La Jolla Village Dr, San Diego, Calif 92161 (Dr. Barrett-Connor).
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