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An Unusual Manifestation of Renal Osteodystrophy

Alvin Sellers, MD; Alan C. Winfield, MD; Shaul G. Massry, MD

Arch Intern Med. 1973;131(5):727-728.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Roentgenographic evidence of bone disease in patients with chronic renal failure is being observed with increasing frequency as the lives of such patients are prolonged with maintenance hemodialysis.¹ The type and severity of bone disease in patients with renal failure are probably related to the degree and duration of renal insufficiency and may be influenced by the dietary habits of the patients.^1-4 Although both the genesis and pathological lesions of renal osteodystrophy are complex, the predominant osseous abnormality in patients treated with maintenance hemodialysis is markedly enhanced bone resorption⁵ induced by the high levels of circulating parathyroid hormone.⁶ The roentgenographic manifestations of secondary hyperparathyroidism are granularity and mottling of the skull, subperiosteal bone resorption of the phalanges, distal end of ulna and femur, and upper end of tibia, and resorption of the lateral ends of the clavicles.⁷ Occasionally, brown tumors and cysts may be seen. . . . [Full Text PDF of this Article]

Author Affiliations

From the Renal and Hypertension Service and departments of medicine and radiology, Cedars-Sinai Medical Center, and the Department of Medicine, UCLA School of Medicine, Los Angeles.

Footnotes

Reprint requests to 8720 Beverly Blvd, Los Angeles 90048 (Dr. Massry).

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