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Steroid-Responsive Hypercalcemia in Disseminated Bone Tuberculosis
MAJ Sidney S. Braman, MC;
MAJ Allan L. Goldman, MC;
MAJ Marvin I. Schwarz, MC
Arch Intern Med. 1973;132(2):269-271.
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There are many established causes of hypercalcemia. These include hyperparathyroidism, malignancies with or without bony metastases, sarcoidosis, bone atrophy, thyroid dysfunction, Addison disease, vitamin D intoxication, and the milk-alkali syndrome.1 Hypercalcemia with disseminated bone tuberculosis is extremely rare. However, a case of miliary tuberculosis in which symptomatic hypercalcemia developed was reversed by corticosteroid therapy.
Patient Summary
A 28-year-old officer began serving a tour of duty in the Republic of Vietnam in September 1969. In March 1970, the patient noted the onset of fever, chills, nausea, and vomiting. A chest roentgenogram was normal, and peripheral blood smears revealed falciparum malaria. Despite several courses of antimalarial therapy and clearing of the parasitemia, fever persisted; and in late May, he was transferred to Walter Reed General Hospital.
On admission, the patient complained of weakness, anorexia, and had a 9.04-kg weight loss. He was a thin, chronically ill appearing, white man in no
. . . [Full Text PDF of this Article]
Author Affiliations
USA; USA; USA; Washington, DC
From the Department of Medicine, Division of Pulmonary and Communicable Disease, Walter Reed General Hospital, and Walter Reed Army Institute of Research, Walter Reed Army Medical Center, Washington, DC.
Footnotes
Received for publication April 28, 1972; accepted May 16.
Reprint requests to OSC#559, Walter Reed General Hospital, Washington, DC 20012 (MAJ Goldman).
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