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Henry D. Royal, MD; John B. Shields, MD; Robert M. Donati, MD

Arch Intern Med. 1975;135(11):1502-1505.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The pathophysiology of pulmonary edema has been extensively studied and reviewed.¹ Predominantly unilateral pulmonary edema has been reported infrequently as a result of patient positioning,^2,3 left ventricular failure,^4,5 increased intracranial pressure,⁶ preexisting lung disease,⁷ congenital heart disease,^8,9 rapid reexpansion of an ipsilateral pneumothorax,^10-14 or reexpansion of a contralateral pneumothorax after acute left-sided heart failure.¹⁵ In contrast, misplacement of central venous catheters is frequent, occurring in 25% to 38% of the cases in which the catheter was believed to be properly placed.^16-19 To our knowledge, misplacement of central venous monitoring catheters into a pulmonary artery has not been reported previously. We have observed two catheters in this aberrant position. In addition, in the case reported here, unilateral pulmonary edema developed following placement of the "central venous" catheter in the ipsilateral pulmonary artery. Experiments in dogs to explore possible mechanisms of pulmonary edema following catheter placement in the pulmonary artery are also . . . [Full Text PDF of this Article]

Author Affiliations

Footnotes

Reprint requests to 593 Eddy St, Providence, RI 02903 (Dr. Royal).

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