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Tricuspid Regurgitation Following Inferior Myocardial Infarction
Russell G. McAllister, Jr, MD;
Gottlieb C. Friesinger, MD;
Bruce C. Sinclair-Smith, MD
Arch Intern Med. 1976;136(1):95-99.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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Isolated tricuspid incompetence is uncommon and is usually tolerated well.1-3 It has been reported after trauma,1-4 with papillary muscle rupture after myocardial infarction,5 after bacterial endocarditis,6,7 after rheumatic fever,8 and as a congenital anomaly.9 In addition, incompetence of the valve after dilatation of the annulus may result from pulmonary hypertension and subsequent severe right ventricular failure.10 In this report, we present two patients with tricuspid insufficiency occurring after inferior wall myocardial infarction and possibly caused by right ventricular papillary muscle dysfunction, a syndrome not previously recognized.11
PATIENT SUMMARIES
PATIENT 1.—
A 68-year-old retired farmer was admitted to Vanderbilt University Hospital because of persistent ascites. He had been well until four years earlier, when he suffered an inferior wall myocardial infarction. Two years after an uneventful recovery, he noted abdominal swelling; this subsequently proved refractory to outpatient therapy with digitalis, sodium restriction, and
. . . [Full Text PDF of this Article]
Author Affiliations
From the Division of Cardiology, University of Kentucky Medical Center, Lexington, Ky (Dr McAllister), and the Division of Cardiology, Vanderbilt University Hospital, Nashville, Tenn (Drs Friesinger and Sinclair-Smith).
Footnotes
Received for publication May 9, 1974; accepted April 25, 1975.
Reprint requests to Division of Cardiology, University of Kentucky Medical Center, Veterans Administration Hospital, Lexington, KY 40506 (Dr McAllister).
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