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Jeffrey Passer, MD

Arch Intern Med. 1976;136(4):462-466.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Symptomatic hypocalcemia and hypomagnesemia, both responsive to replenishment of magnesium, occur in chronic alcoholism,¹ malabsorption,² experimentally induced magnesium deficiency,³ primary magnesium deficiency,⁴ and severe malnutrition.^3,5 The cause of hypocalcemia in these conditions is unknown. Postulated mechanisms include decreased secretion or defective synthesis of parathyroid hormone^6,7 or peripheral resistance to parathyroid hormone.^4,8

I will describe two patients with hypocalcemia and hypomagnesemia who also had tetany, incomplete distal renal tubular acidosis, and increased concentrations of serum parathyroid hormone, all of which conditions responded to magnesium replenishment.

PATIENT SUMMARIES

PATIENT 1.—

In 1954, a female patient developed diarrhea and abdominal pain. She was found to have regional enteritis, and a portion of small bowel was resected. In 1962, another portion of small bowel was resected because of acute intestinal obstruction. Two months later, the patient developed tetany and hypocalcemia and was treated with orally and intravenously . . . [Full Text PDF of this Article]

Author Affiliations
From the Department of Internal Medicine, Bishop Clarkson Memorial Hospital, University of Nebraska College of Medicine, Omaha.

Footnotes
Received for publication Jan 7, 1975; accepted Sept 19.

Reprint requests to 530 Doctors' Bldg, 4239 Farnam St, Omaha, NB 68131.

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