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[ill] Roy First, MD; Molly Ettenger, MD; Michael Robson, MD; [ill]ictor E. Pollak, MD; Boon Seng Ooi, MD; Martin Goldberg, MD

Arch Intern Med. 1984;144(11):2233-2238.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

[ill] ROY FIRST, MD, Associate Professor of Medicine: Sud[ill]en deterioration in the clinical condition of a patient with [ill]hronic renal insufficiency may occur frequently as a result [ill]f a superimposed acute illness with consequent deteriora[ill]ion in renal function. It is important to identify and treat [ill]ny condition that aggravates the underlying renal disease [ill] has adverse effects on renal function.¹

Table 1 illustrates those conditions causing acute deterio[ill]tion of renal function in the patient with azotemia or [ill]emia.¹ Reduced renal perfusion commonly results from [ill]xtracellular fluid volume depletion. The use of potent [ill]iuretic agents, eg, furosemide or ethacrynic acid, coupled [ill]ith the injudicious restriction of dietary sodium, is a [ill]mmon cause of salt and water depletion, as are gastroin[ill]stinal (GI) losses of sodium and water by vomiting, [ill]arrhea, or both from intercurrent GI disturbances or [ill]om the uremia per se. Subtle dehydration also may result [ill]hen the patient, who is . . . [Full Text PDF of this Article]

Author Affiliations
From the Division of Nephrology, University of Cincinnati Medical [ill]nter.

Footnotes
Accepted for publication May 7, 1984.

Reprint requests to the Division of Nephrology, University of Cincinnati [ill]dical Center, 5363 MSB, 231 Bethesda Ave (585), Cincinnati, OH 45267-[ill]5 (Dr First).

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