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William B. Kannel, MD

Arch Intern Med. 1996;156(11):1138-1139.

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THERE IS now incontrovertible evidence that dyslipidemia is fundamental to the atherosclerotic process and that correction of the disorder slows atherogenesis, stabilizes lesions, and reduces the morbidity and mortality of coronary heart disease (CHD).^1,2 There is also ample evidence that faulty nutrition, ie, a diet consisting of too much saturated fat and cholesterol, too little fiber, and too many calories for the level of activity, is the chief determinant of blood lipid values in the general population.³ The average serum total cholesterol level of the general population has declined over the years.⁴ In the Framingham Study cohort it has declined each calendar decade, almost certainly as a result of changes in fat and cholesterol intake.⁵ Migrants from low to high CHD areas of the world raise their cholesterol level, and along with this, their CHD mortality rate.⁶ Thus, for the general population, attention to nutrition . . . [Full Text PDF of this Article]

Author Affiliations
Framingham Heart Study Boston University School of Medicine 5 Thurber St Framingham, MA 01701

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