This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

L. H. NEWBURGH, M.D.

Arch Intern Med. 1919;24(4):359-377.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

For nearly a century the etiology of Bright's disease has been the subject of much medical thought and investigation. But in spite of this, the clinician is still unable to find a cause for the disease in the great majority of cases seen by him. The present investigation was undertaken with the hope of throwing some additional light on this difficult subject.

The chief business of the kidneys is to rid the organism of the end products of protein metabolism. This task is accomplished to a considerable extent by active secretion on the part of certain portions of the tubular epithelium. These cells will not only remove normal waste, but they will also remove from the blood stream substances which are foreign to it even though they may be severely injured by the poisons passing through them. Thus investigators¹ have shown that when the kidney eliminates such substances as mercury, . . . [Full Text PDF of this Article]

Author Affiliations
ANN ARBOR, MICH.

From the Department of Internal Medicine, University of Michigan.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?