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THE ALTERATIONS WITH SOME OBSERVATIONS ON THE ETIOLOGY OF THE DISEASE

CHESTER M. JONES, M.D.; WILLIAM B. CASTLE, M.D.; HENRY B. MULHOLLAND, M.D.; FRANCIS BAILEY, B.S.

Arch Intern Med. 1925;35(3):315-336.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

A brief preliminary report¹ has already been made of the findings obtained in a study of pancreatic and hepatic activity in cases of diabetes mellitus. It is the purpose of this paper to present these findings in detail and, so far as our findings seem to bear on them, to comment on certain considerations in this disease.

REVIEW OF THE LITERATURE

Although the recent work of Banting² and his collaborators has contributed an invaluable addition to our methods of treating diabetes mellitus, there still remains much to be desired in our knowledge of the actual etiology of this disease. That a deficiency of the internal secretion of the pancreas is the immediate cause of the glycosuria is incontestably proved, but the extent to which other organic functions are involved still remains a question. According to Allen,³ in most, if not in all, cases of diabetes mellitus there must have been . . . [Full Text PDF of this Article]

Author Affiliations
BOSTON

From the Medical Services of the Massachusetts General Hospital, and the New England Deaconess Hospital.

Footnotes
This paper is No. 28 of a series of studies in metabolism from the Harvard Medical School and allied hospitals. The expenses of this investigation have been defrayed in part by a grant from the Proctor Fund of the Harvard Medical School for the Study of Chronic Disease.

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