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HERBERT W. SCHMITZ, M.D.; EDWARD L. ROHDENBURG, B.S.; VICTOR C. MYERS, Ph.D.

Arch Intern Med. 1926;37(2):233-243.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

It has been recognized for several years that the terminal stages of nephritis are accompanied by an increase in the inorganic phosphorus of the blood serum and some decrease in the calcium.

In 1915 Greenwald¹ reported observations on the acid-soluble (largely inorganic) and lipoid phosphorus of human blood serum. He observed that normally the acid-soluble phosphorus as P varied between 2 and 6 mg. per hundred cubic centimeters, but that in severe nephritis it might be considerably increased. A year later Marriott and Howland² confirmed these observations and pointed out that the retention of (acid) phosphate would seem to be sufficient to account for the degree of acidosis observed. They found further in most of their cases a marked reduction in the calcium of the serum. They state:

What influence this low calcium content may have on the production of such symptoms as convulsions and hemorrhages can only be suggested. . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK; IOWA CITY

From the Department of Biochemistry, New York Post-Graduate Medical School and Hospital.

Footnotes
Medical fellow of the National Research Council during part of this study.

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