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D. T. BARRY, M.D.

Arch Intern Med. 1927;40(4):446-470.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Previous workers on experimentally induced valvular incompetence in the heart have been almost exclusively occupied with lesions of the mitral valve and their limited aspects, namely, the period of the cycle in which the backflow into the auricle actually occurs, the pressure changes set up in the auricle and ventricle and the factors concerned in the compensation of the defect. Other sides of the problem which are at least as important as those mentioned, have scarcely been considered up to now, such as actual measurement of the output from the heart during the course of a lesion, which is the best criterion of compensation; nor has a contrast of the function with mitral lesions been made with that done in the condition of tricuspid defect. The present communication deals with the results of recent work on these problems and is based on a long series of experiments on dogs, operations . . . [Full Text PDF of this Article]

Author Affiliations
CORK, IRELAND

From the Physiological Laboratory, University College, Cork.

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