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RUSSELL L. CECIL, M.D.; EDITH E. NICHOLLS, M.D.; WENDELL J. STAINSBY, M.D.

Arch Intern Med. 1929;43(5):571-605.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

This study deals with the etiology of a form of chronic arthritis which has been variously referred to as rheumatoid arthritis, arthritis deformans or chronic infectious arthritis. Goldthwaite¹ and his school call it atrophic arthritis. It may be defined as a chronic inflammatory condition of the joints and periarticular tissues, characterized in the earlier stages by migratory swelling and stiffness of the joints, and in the later stages by more or less deformity and ankylosis. In the great majority of cases, several joints are involved; indeed, one of the most striking features of the disease is its tendency, if unchecked, to progress and finally to involve nearly every joint in the body.

The etiology of chronic deforming arthritis has long been a subject for debate. Perhaps no disease in the whole realm of internal medicine has been more prolific of hypotheses and theories. It has long been recognized that there . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Cornell Clinic and the Second (Cornell) Medical Division of Bellevue Hospital.

Footnotes
Submitted for publication, Feb. 15, 1929.

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