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BACTERIOLOGY OF AFFECTED TISSUES

HARRY M. MARGOLIS, M.D.; ANNA H. E. DORSEY, M.S.

Arch Intern Med. 1930;46(1):121-136.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In an attempt to explain the etiology of nonspecific chronic arthritis, there have emerged widely conflicting opinions based, at times, entirely on clinical or laboratory observations. But in view of the lack of definitive conclusions, it seems desirable to correlate our laboratory observations with clinical observations to determine what light may be shed on this rather perplexing problem.

The opinions of most writers in recent years have begun to lean toward the primarily infectious nature of chronic arthritis. Although, admittedly, the modus operandi of such infections has not been clarified, a great amount of reliable data has already accumulated in support of the infectious theory. Clinically, the frequent occurrence of arthritis in the course of acute infections such as scarlet fever, typhoid fever, malta fever, ulcerative colitis, and so forth, is well recognized. The extensive researches of Rosenow¹ and his co-workers² have emphasized the rôle of focal infection in arthritis . . . [Full Text PDF of this Article]

Author Affiliations
Fellow in Medicine; Fellow in Bacteriology, the Mayo Foundation ROCHESTER, MINN.

Footnotes
Submitted for publication, Dec. 1, 1929.

Work done in the Division of Medicine and Division of Experimental Bacteriology under the direction of L. G. Rowntree, P. S. Hench and E. C. Rosenow.

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