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MAX WISHNOFSKY, M.D.; CHARLES S. BYRON, M.D.

Arch Intern Med. 1931;47(5):790-798.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

It has been noted by clinical observers that a disturbance of carbohydrate metabolism exists in hypertension.

In 1910, Neubauer¹ observed the association of hyperglycemia in nephritis with hypertension, and postulated that an overactivity of the suprarenal glands was the causative factor. Hagelberg² made the same observation and accepted Neubauer's theory. Tachau³ also concurred in these observations, but did not attempt to explain the mechanism involved. Bing and Jakobsen⁴ were of the opinion that the hyperglycemia in all cases of hypertension with nephritis could be explained by a complicating condition such as dyspnea, uremia or a cerebral accident.

Hopkins⁵ also observed hyperglycemia in hypertension with nephritis. Hamman and Hirschman⁶ found that cases of high blood pressure, especially when complicated by nephritis, showed a so-called "diabetic" blood sugar curve after the administration of dextrose. Hirsch⁷ noted that in the majority of cases of vascular hypertension without nephritis there was hyperglycemia and an . . . [Full Text PDF of this Article]

Author Affiliations
BROOKLYN

From the Department of Metabolism and Pathology and the Medical Clinic (Service of Dr. S. R. Blatteis) of the Jewish Hospital.

Footnotes
Submitted for publication, Sept. 22, 1930.

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