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MARTIN H. DAWSON, M.D.; MIRIAM OLMSTEAD, A.M.; RALPH H. BOOTS, M.D.

Arch Intern Med. 1932;49(2):173-180.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The clinical evidence pointing to an infective origin of rheumatoid arthritis has led numerous investigators to seek a bacterial agent to which etiologic significance could be ascribed. A wide variety of such agents has been reported by various workers at various times, but the lack of uniformity in the results obtained has served to confuse rather than to clarify the issue. In recent years the widespread interest in rheumatoid arthritis and the recognition of the social and economic importance of this disease have stimulated renewed activity in the search for the etiologic agent.

HISTORICAL

Bacteriologic studies on the blood and tissues in rheumatoid arthritis are so numerous that a complete review of the results obtained is not within the scope of this report. Moreover, a critical review of the literature is difficult, for numerous investigators have presented the results obtained without reference to the technic employed, the kind of cases . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Department of Medicine, College of Physicians and Surgeons of Columbia University, and the Arthritis Clinic of the Presbyterian Hospital (the Arthritis Clinic of the Presbyterian Hospital is supported by the Faulkner Memorial Fund).

Footnotes
Submitted for publication, May 25, 1931.

The Arthritis Clinic of the Presbyterian Hospital has adopted the classification and nomenclature for rheumatic diseases provisionally recommended by the British Ministry of Health and the International League for the Control of Rheumatism. In this classification the term "rheumatoid arthritis" is used to designate that form of chronic multiple arthritis more commonly called "chronic infectious" or "atrophic" in America.

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