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J. PAUL BURGESS, M.D.; HORACE G. SCOTT, M.D.; A. C. IVY, M.D.

Arch Intern Med. 1932;49(3):439-452.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The mechanism concerned in the production of acute dilatation of the stomach is not fully understood. Numerous theories have been advanced to account for it, but none has been entirely adequate. The reason is perhaps to be explained by the fact that this condition may be brought about in several different ways.

When first described by Brinton¹ it was thought to be primarily a passive phenomenon of a purely paralytic nature. Conner² stated: "The absence in almost all cases of obvious obstruction at the pylorus, and the fact that a similar paralytic distention of the intestines is a not uncommon phenomenon after abdominal operations and in peritonitis served to support this view as has also the experimental work of Carion and Hallion."³ The latter investigators said that "after resection of both nerves (vagi) above the diaphragm we have seen produced in the dog an enormous dilatation of . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Department of Physiology and Pharmacology, Northwestern University Medical School.

Footnotes
Submitted for publication, June 29, 1931.

This investigation has been aided by a grant from the Josiah Macy, Jr., Foundation.

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