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LEO M. ZIMMERMAN, M.D.; SAMUEL SOSKIN, M.D.

Arch Intern Med. 1932;49(4):663-665.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In another study,¹ the results of ligation of the parotid duct on the carbohydrate tolerance of normal dogs were reported. In these animals, it was found that the tolerance for dextrose, as determined by the blood sugar curve following the intravenous injection of dextrose solution, was definitely increased by ligation of the parotid ducts. As a step in the study of the mechanism of this action, the effect of ligation of the parotid duct was determined in a series of totally depancreatized dogs. Mansfeld and Schmidt² found that after partial extirpation of the pancreas (Sandmeyer diabetes) the resulting disturbances in carbohydrate metabolism were greatly relieved by ligation of the parotid ducts. In one animal in which the entire pancreas had been removed they were unable to detect any improvement following ligation of the parotid duct. Seelig³ found that ligation of Stenson's ducts did not prevent animals from . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Metabolic Laboratory of the Department of Physiology, Nelson Morris Institute for Medical Research, Michael Reese Hospital.

Footnotes
Submitted for publication, June 24, 1931.

Under a grant from the Max Pam Fund for Metabolic Research.

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