This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

FRANKLIN R. NUZUM, M.D.; ALBERT H. ELLIOT, M.D.

Arch Intern Med. 1932;49(6):1007-1018.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Much experimental and pathologic evidence has accumulated to indicate that the pain of angina pectoris is due to disturbance of the coronary circulation. The ideal therapeutic measure, then, would be one that insures at all times an adequate flow through the coronary arteries. Of the various drugs used in the treatment of this disease, those which are known to dilate the coronary arteries have received the widest acceptance. For relief from the attack, the nitrites are invaluable, not only because they reduce the systemic blood pressure and thus relieve in part the burden imposed on the heart muscle, but also because in all probability they decrease myocardial ischemia. Unfortunately, their effect is transitory. For the prevention of attacks, another class of drugs which act in much the same manner, namely, the purine derivatives (theobromine, theophylline, etc.) have proved of value. However, these drugs fall short of the ideal because they . . . [Full Text PDF of this Article]

Author Affiliations
SANTA BARBARA, CALIF.

From the laboratories of the Cottage Hospital.

Footnotes
Submitted for publication, Oct. 12, 1931.

Read before the Section on Practice of Medicine at the Eighty-Second Annual Session of the American Medical Association, Philadelphia, June 10, 1931.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?