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J. LISLE WILLIAMS, M.D.; GEORGE F. DICK, M.D.

Arch Intern Med. 1932;50(6):801-818.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Transient glycosuria has been noted in various infectious diseases. Hibbard and Morrissey¹ found it in diphtheria. Cammidge² stated that it occurs in diphtheria, scarlet fever, typhoid fever, influenza, appendicitis, measles and infections with suppuration. Buhl³ described its occurrence in Asiatic cholera. Castellani and Willemore⁴ and Harrison⁵ have found that it may be present in malaria. Cammidge⁶ and Higginson⁷ have designated as "sapraemic glycosuria" that accompanying carbuncle or gangrene. This glycosuria is accompanied by a hyperglycemia according to Hollinger.⁸ A lowered dextrose tolerance was demonstrated by Hamman and Hirschman⁹ in lobar pneumonia and acute tonsillitis. Olmsted and Gay¹⁰ found abnormal blood sugar curves in many conditions including those resulting from acute infectious toxins. Tisdall, Drake and Brown¹¹ noted a derangement of the carbohydrate metabolism in infants with acute infectious diarrhea. In the study of various infectious diseases Labbé and Boulin¹² found an increase in the height of the . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the John McCormick Institute for Infectious Diseases.

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