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RICHARD T. BEEBE, M.D.; M. M. WINTROBE, M.D.

Arch Intern Med. 1933;52(3):464-470.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The reports of cases of idiopathic hypochromic anemia continue to increase, but controversy exists, nevertheless, as to whether or not this condition can be accepted as a clinical entity. Some authors¹ think of this anemia as an atypical form of pernicious anemia. Others consider that it is a form of anemia resulting from undiscovered chronic loss of blood. Opinions² have been expressed that the anemia is the result of dietary deficiencies or that it is related to chlorosis, once so common in young girls. The view most generally held, however, is that the anemia and the clinical picture are the result of some deficiency in gastric secretion³ or the consequence of an inability of the gastrointestinal tract to absorb from the food substances necessary for the formation of blood.⁴

In 1929, Castle⁵ showed that when beef steak properly digested with normal gastric juice was fed . . . [Full Text PDF of this Article]

Author Affiliations
ALBANY, N. Y.; BALTIMORE

From the Medical Clinic, the School of Medicine, Johns Hopkins University and Hospital.

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