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ARTHUR M. FISHBERG, M.D.; WILLIAM M. HITZIG, M.D.; FREDERICK H. KING, M.D.

Arch Intern Med. 1934;54(6):997-1019.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The clinical evidences of circulatory failure in the first days of myocardial infarction often differ conspicuously from the manifestations of decompensation in other diseases of the heart. In many instances the absence of orthopnea is striking; the patient evinces no distress when lying flat in bed despite the fact that grayish cyanosis and cold extremities testify to the impairment of the peripheral circulation. Moreover, inspection of the cervical and other superficial veins reveals that they are largely collapsed. These observations are in sharp contrast to those which one is accustomed to encounter when equally severe retardation of the peripheral circulation occurs in such conditions as, for example, mitral stenosis. In that case the patient is orthopneic, and the superficial veins are engorged. The picture in myocardial infarction is obviously the one which is familiar to every physician under the name of shock and which results from disturbances in the periphery . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Medical Services and the Laboratories of the Mount Sinai Hospital.

Footnotes
Eugene Meyer Fellow. Aided by a grant to the Emanuel Libman Fellowship Fund in memory of Adele Schiff.

Emanuel Libman Fellow.

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