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WILLIAM M. HITZIG, M.D.; FREDERICK H. KING, M.D.; ARTHUR M. FISHBERG, M.D.

Arch Intern Med. 1935;55(1):112-120.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Increased attention has been devoted of late to the old clinical concept of left ventricular failure. Excellent surveys have been reported by White¹ and by Weiss and Robb.² The clinical manifestations are those resulting from pulmonary engorgement, and include various combinations of the following: exertional dyspnea, cardiac asthma, orthopnea, cyanosis, cough, blood-streaked sputum, physical signs and roentgen evidence of pulmonary engorgement, diminution in vital capacity, pulmonary edema, dilatation of the left ventricle and auricle, accentuation of the pulmonic second sound, the systolic murmur of relative mitral insufficiency, gallop rhythm, fall in arterial pressure and, rarely, alternation of the pulse. On the other hand, engorgement of the systemic veins, swelling of the liver, dependent edema and other manifestations of insufficiency of the right side of the heart are absent.

The characteristic clinical picture of isolated failure of the left side of the heart is encountered only when the functional . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the medical service and laboratories of the Mount Sinai Hospital.

Footnotes
Eugene Meyer Jr. Fellow in Pathology.

The investigation was aided by a grant to the Emanuel Libman Fellowship Fund in memory of Adele Schiff.

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